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Sep 17 15 4:20 AM

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After dealing with whatever protozoan I have for several years, it very well could be Protomyxzoa Rheu.  My opinon has changed.  I now believe that PR is only a small piece of a big puzzle.  In people with normal immune function, it causes little or no problems or issues.  In people with multiple pathogens, it's a huge problem because of the biofilms.  I've conducted multiple experiments with myself to see how I responded.  I learned really quick what I could handle and what I couldn't handle.  I do believe that in increases my body's ability to accumulate metals, especially aluminum.  I feel that over time, it depletes polyvalent cations that are necessary for normal cellular functioning.  Copper, Selenium, Manganese, and Molybdenum are the ones I believe I am deficient of.  These are substrates for important protein transfer through cellular membranes and also catalysts for methylation pathway reactions.  When I increase my intake or supplement with these elements, my inflammation increases.  My theory is that we deplete the elements and that causes the down regulation of our immune response.  Our limitations in treatment are how much inflammation and cellular debris we can handle and eliminate. 

I believe that my protozoan has a structure similar to sphingomyelin and are incorporated into the cellular structure of the myelin sheaths around muscles and in synovial membranes.  Catechins negatively affect my protozoans and their chemical structure is very similar.  Without exercising, there is no way to kill/liberate them from their location.  I can herx just from mild exercise if I use muscles I haven't used very often. 
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Sep 18 15 3:40 AM

Have you tried to eliminate the biofilm? Lactoferrin or lactoferrin with xylitol is a powerful tool. I have a post on this. Also just made a post about Kra Chai, apparently it has anti biofilm properties though I do not know how it would compare to the lactoferrin.  Here is what they say about it:5.1. Inhibition of Biofilm Formation by Oral PathogensBiofilm formation on teeth surfaces is caused by multiple species of oral bacteria, the primary colonisers being mutant Streptococci [72]. Biofilm formation is associated with several acute and chronic infections such as dental caries, gingivitis, and periodontitis and potentially contributes to antibiotic treatment failure againstStreptococcus pyogenes [48]. In 2008, Limsuwan and Voravuthikunchai demonstrated that the extracts from B. rotunda, Eleutherine americana, and Rhodomyrtus tomentosa exhibited antibiofilm activity towards S. pyogenesat subinhibitory concentrations (1/32–1/2 MIC) for E. americana (7.81–125 μg/mL), R. tomentosa(0.24–7.81 μg/mL), and B. rotunda (1/2 MIC of 7.8 μg/mL). Antiquorum sensing test revealed that B. rotundashowed no inhibition activity, while R. tomentosa displayed strongest antiquorum effect followed by E. americana with moderate effect. However, microbial adhesion to hydrocarbon assay showed no changes in the cell-surface hydrophobicity of treated bacteria [48].The following year, Yanti et al. [49] reported the anti-biofilm property of B. rotunda extracted panduratin A, which was found to prevent and reduce the spread of multispecies oral bacteria in human mouth. The MIC of panduratin A was determined using the Clinical and Laboratory Standards Institute (CLSI) broth microdilution assay. Mucin-mixed panduratin A at concentrations between 0.5 and 40 μg/mL was coated on 96-well plates, followed by inoculation of three multispecies bacteria, Streptococcus mutans, Streptococcus sanguis, andActinomyces viscosus, and incubated overnight at 37°C to allow biofilm formation. Biofilm reduction effect was determined by further treating the bacteria with different concentrations of panduratin A (0.2–10 μg/mL) for up to 60 mins. Panduratin A exhibited bacteria reduction effect at MIC of 1 μg/mL and bactericidal effect against multispecies planktonic cells at 2X MIC, 8 hours after treatment. Reduction of biofilm formation was >50% at 8X MIC, whereas mass reduction of biofilm was observed within 15 mins at a concentration of 10 μg/mL [49]. These results suggested that panduratin A can potentially be used to prevent colonisation of multispecies bacteria, under a dose-dependent manner, and that its effect is equal to commercially available synthetic drugs such as chlorhexidine gluconate [49].

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